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Advanced Cardiac Life Support

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Advanced Cardiac Life Support

Cardiac Arrest Management/AED

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New York State Protocols Update 2006 Including AHA changes.

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BRADY Chris Fraser Introduction to High- Performance CPR.

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Final version 1, RESUSCITATION OUTCOMES CONSORTIUM C ontinuous C hest C ompressions Trial Final version 1,

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CARDIOPULMONARY RESUSCITATION

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Electrical Therapies Isfahan University Of Medical Sciences

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European Resuscitation Council

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Project: Ghana Emergency Medicine Collaborative Document Title: ACLS Overview: Pulseless Arrest Author(s): Rockefeller Oteng (University of Michigan),

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advanced cardiac life support acls

Advanced Cardiac Life Support (ACLS)

Dec 10, 2012

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Advanced Cardiac Life Support (ACLS). By: Diana Blum MSN Metropolitan Community College Nursing 2150. STABLE.  These patients generally have an EKG rhythm that is undesirable. their vitals signs are stable they have no complaints such as, shortness of breath, chest pain or confusion.

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Advanced Cardiac Life Support (ACLS) By: Diana Blum MSN Metropolitan Community College Nursing 2150

STABLE •  These patients generally have an EKG rhythm that is undesirable. • their vitals signs are stable • they have no complaints such as, shortness of breath, chest pain or confusion. • if rhythm untreated the patient may become ____________.

UNSTABLE  • These patients also have an EKG rhythm that is undesirable. • vital signs are not stable! • Other sign and symptoms: low blood pressure, shortness of breath, chest pain or confusion. • if the rhythm is not treated the patient may die.. • BE AGGRESSIVE in approach in unstable patients. • You should always do CPR until code cart is available. Rhythms Too fast; like ventricular tachycardia, or ventricular fibrillation we defibrillate. Absent, as in asystole we pace with a Trans Cutaneous Pacing patches.

DEAD • These patients also have an EKG rhythm that is undesirable. • vital signs are absent! They have no pulse! • Your first thought for intervention is SHOCK EM! Especially if witness going down. • Step 2 CPR. ---new protocol is compressions compressions compressions! • The last intervention in order is MEDICINE. • "all dead people get epinephrine, the deader they are, the more epinephrine they get!" • American Heart studies show that the sooner electrical intervention is introduced, the better the outcome for survival! • Your second intervention is CPR. Think of CPR as your bridge and time-buyer. • Good CPR keeps the vital organs per fused until your electrical and drugs can do their job. • Always make good CPR a priority.

Primary Survey • Airway: Open airway, look, listen, and feel for breathing • Breathing: If not breathing slowly give 2 rescue breaths. If breaths go in continue to next step. • Circulation: check pulse 5-10 seconds • Defibrillation: Search for a shockable rhythm like vtach/vfib

Adult ACLS Secondary Survey ABCDs (abbreviated) • Airway: Intubate if not breathing. Assess bilateral breath sounds for proper tube placement. • Breathing: Provide positive pressure ventilations with 100% O2. • Circulation: If no pulse continue CPR, obtain IV access, give proper medications. • Differential Diagnosis: Attempt to identify treatable causes for the problem.

http://www.youtube.com/watch?v=tVHJq9op5cw&feature=relmfu

Pulseless Electrical Activity, or PEA • This is a condition where you have some electrical activity but not mechanical activity. • AKA: no pulse is present. • You can have a normal sinus rhythm, but if there is no pulse, the condition is called PEA. • If you have a patient with the condition of PEA, and the rhythm is a slow wide ventricular rhythm, you may want to try TCP.

PEA • Problem search..Treat accordingly. (see differential diagnosis table) Epinephrine 1 mg IV/IO q3-5 min. Or vasopressin 40 U IV/IO, once, in place of the 1st or 2nd dose of epi. Atropine 1 mg IV/IO q3-5 min. (3mg max.)

ELECTRICAL! • If the rhythm is too fast, the goal is to slow it down and convert it • use synchronized cardioversion. • If too slow the goal is to speed it up, • use external transcutaneous pacing or TCP. • “ how do I know when to pace, defibrillate, or use synchronized cardioversion?" • HINT: D=Deceased, • only defibrillate fast rhythms! • look at suspected asystole in more that one ekg lead, to confirm asystole.

Bradycardia • HR (<60bpm) or relative (slower rate than expected) bradycardia with circulatory compromise. Start the Secondary ABCDs • Pacing:Immediately prepare for transcutaneous pacing related to bradycardia (especially high-degree blocks) or if atropine failed to increase rate. • Always Atropine1st-line drug, 0.5 mg IV/IO q3-5 min. (max. 3mg) • Ends: Epinephrine2-10 µg/min2nd-line drugs to consider if atropine and/or TCP are ineffective.. • Danger: Dopamine2-10 µg/kg/min • *pacing may not work every time with brady arrhythmias. If the above measures do not improve circulatory stability the bradycardia may be from other issues, think differential diagnosis! (Refer to slide 10)

Cardioversion • Synchronized Electrical Cardioversion • the following mnemonic directs preparations for synchronized electrical cardioversion of unstable tachycardia with fast rate (do not delay shocking if seriously unstable) • Oh O2 Saturation monitor • Say Suctioning equipment • It IV line • Isn't Intubation equipment • So Sedation and possibly analgesics **Synchronized Electrical Cardioversion *Energy Levels:The initial synchronized shock is 100J monophasic (50J for SVT/A-Flutter) with increasing energy, i.e., 200J, 300J, 360J, if successive shocks are needed.

Adult Cardiac Arrest

1st Start CPR • Is the rhythm shockable? Yes or No • If shockable (VF/VT)? Yes or NO • If not shockable(Asystole)? Yes or NO • If VF/VT • Shock • CPR x 2 minutes • Get IV/IO access • Reanalyze (shockable??) • Yes • Shock then CPR x 2minutes and or epinephrine/capnography • NO • CPRx 2 minutes, epinephrine/ Airway • Repeat steps as needed • Asystole • CPR x 2 minutes, , epinephrine/ Airway • Reanalyze • Shockable • Yes • Shock cpr epinephrine airway • No • CPR x 2 minutes, treat causes

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AHA Guidelines ACLS

Aha guidelines acls – powerpoint ppt presentation.

  • Know your protocols
  • Know the standards (algorithms)
  • Be flexible
  • Your patient probably did not read the ACLS manual
  • ILCOR considered over 350 literature based reviews of specific resuscitation related questions
  • There were more questions than answers
  • Change is bad
  • Every time guidelines change, basic skills must be relearned
  • Changes based on weak data may undermine the integrity of the process
  • Guidelines 2000 Epinephrine Class indeterminate
  • Vasopressin Class 2b
  • Guidelines 2005 Epinephrine Class 2b
  • Vasopressin Class indeterminate
  • Change is good
  • New skills save lives
  • New data demands new recommendations
  • Consensus may not be the right thing if there is definitive data.
  • When is consensus best?
  • Consensus works when it takes something everybody knows and makes it something everybody agrees to do.
  • Quote from Developing Consensus in Emergency Medicine Information Technology AEM 11/04
  • Reviewing ACLS is impossible without an overview of CPR
  • CPR will be taught as an integral part of every ACLS course
  • The goal is to use CPR to increase the efficacy of defibrillation to achieve a perfusing rhythm
  • Good Data (Aufderheide, Abella and others) that we do too many ventilations.
  • Universal agreement that we need to decrease ventilations.
  • NO data directly comparing compression ventilation ratios
  • Most wanted to see a unified pediatric/adult ratio
  • All courses (BLS, ACLS, PALS) will teach CPR as part of core course
  • Mega-codes will incorporate real time CPR with retesting for improper performance
  • Rapid rate with full chest recoil is emphasized
  • Rate of 302 compression to ventilation with BVM
  • 100 compressions unsynchronized to 8-10 ventilations controlled airway
  • Research indicates- 2 minutes of CPR prior to first defibrillation for a non witnessed cardiac arrest
  • Prime the ATP pump
  • Otherwise VFIB will go to
  • Asystole or PEA
  • Dont interrupt compressions
  • Stopping compressions for 4 seconds
  • Reduces Coronary Perfusion Pressure (CPP) to Zero.
  • Studies show we stop TOO long.
  • Every other intervention ( except defibrillation) in cardiac arrests should be
  • Considered an adjunct to chest compressions.
  • Rotation of the cardiac compressor should be every 2 minutes
  • Bag-Mask ventilation is emphasized
  • ..all healthcare providers should be trained in delivering effective oxygenation and ventilation with a bag and mask.
  • Definitive airway may be delayed
  • Rescuers may defer insertion of an advanced airway until the patient fails to respond to initial CPR and defibrillation attempts
  • Stacked shocks are eliminated. Start with maximum energy setting
  • There is no data to support any number of stacked shocks
  • Biphasic defibs have increased conversion efficacy (gt90 in electrical phase)
  • Delays in time between last compression and shock significantly change shock efficacy
  • PEA or asystole are common for a brief period post shock
  • Ischemic myocardium is likely to develop recurrent ventricular fibrillation
  • CPR has not been shown to be detrimental even in the patient with a pulse.
  • Exactly that
  • Look at the rhythm.
  • If organized do pulse check
  • If not resume CPR without pulse check
  • Route of administration
  • Intraosseous is now a recommended route of administration (IIa)
  • Endotracheal administration ONLY if IV/IO cannot be established (still 2-2.5 times IV dose)
  • Vasopressors
  • Epinephrine Upgraded from class indeterminate to class 2b.
  • Vasopressin Downgraded from 2b to class indeterminate
  • Epinephrine is recommended in ALL pulseless arrests
  • Vasopressin may replace 1st or 2nd of epinephrine. Timing does NOT change
  • One large study showed no difference in survival between epinephrine and vasopressin and a trend to better ROSC with both
  • Epinephrine is the established vasopressor
  • Because vasopressin effects have not been shown to differ from those of epinephrine in cardiac arrest one dose of vasopressin may replace..
  • CONSIDER Antiarrhythmics
  • Amiodarone 300 mg IV/IO once then consider additional 150 mg IV/IO once.
  • Lidocaine 1-1.5 mg/kg first dose then 0.5-0.75 mg/kg maximum 3 doses or 3mg/kg
  • Consider Magnesium 1-2 gm for torsades de pointes
  • There is no evidence that any antiarrhythmic drug increases survival to hospital discharge.
  • V FIB - PULSELESS V TACH
  • Initial Intervention
  • Identify absence of pulse and respirations.
  • CPR for 2 minutes if unwitnessed arrest.
  • Defibrillate
  • Witnessed Arrest
  • EKG or paddles for "Quick Look." Determine V fib or V tach.
  • Defibrillate 200 J (Biphasic defibrillator) 360J (Monophasic defibrillator)
  • Children 2 J/kg first shock 4 J/kg subsequent.
  • Unwitnessed Arrest
  • 2 minutes of CPR then defibrillate as per above
  • I.V. balanced salt solution, TKO. ET Tube, 100 O2.(maintain EtCO2 of 30-40)
  • V FIB - PULSELESS V TACH (cont.)
  • Drug Therapy - (CPR for 2 minutes then defibrillate after each drug given).
  • Initial drug (adults only)
  • Vasopressin 40 units IV, single dose one time only
  • After 3-5 minutes
  • Epinephrine 1.0 mg IV or IO. Repeat every 3-5 minutes prn
  • Children 0.01mg/kg initial and subsequent doses
  • If V-Fib persists, administer antiarrhythmic (If ET CO2 gt 10)
  • Amiodarone 300 mg IV/IO bolus, repeat 150 mg IV bolus in 3-5 minutes prn. Children 5 mg/kg IV/IO bolus
  • If Amiodarone contraindicated Lidocaine 1.5 mg/kg IV/IO bolus, repeat 0.75 mg/kg bolus in 3-5 minutes prn max 3 mg/kg. Children 1 mg/kg
  • If multifocal WCT (Torsades) or Magnesium deficiency suspected
  • Magnesium Sulfate 2 gm bolus I.V. (dilute in 50cc D5W wide open) Children 25-50 mg/kg
  • Continuously monitor effectiveness of CPR and oxygenation. May repeat defibrillation current, as appropriate, after each 2 min of CPR (5 cycles).
  • Supraventricular rhythm greater than 60/minute with pulses
  • Utilize an infusion rate of the drug associated with restoration of a stable rhythm. If no antiarrhythmic given treat symptomatically.(Reduce maintenance dose only if impaired liver or heart chf, etc )
  • Bradycardia
  • Atropine 0.5 mg I.V. repeat as needed every 2-3 minutes (3mg total).
  • Epinephrine per protocol
  • Dopamine drip per protocol
  • The recommended dose of atropine for cardiac arrest is 1 mg IV/IO. Maximum 3 mg. (Class indeterminate)
  • No pacing in asystole
  • Several randomized controlled trials failed to show benefit from attempted pacing for asystole. At this time use of pacing for patients in asystolic cardiac arrest is not recommended.
  • CPR for 2 mins
  • Apply EKG Leads. Determine asystole in two leads.
  • I.V. TKO with balanced salt solution
  • ET tube, 100 O2.
  • Electrical Therapy
  • Consider immediate transcutaneous pacemaker if
  • Perfusing Bradycardia converting to asystole during resuscitation
  • Asystole due to Adenocard administration
  • Drug Therapy
  • Epinephrine 1.0 mg I.V. or IO. Repeat every 3-5 minutes prn Children 0.01mg/kg initial and subsequent doses
  • Atropine 1mg I.V. or IO q 3-5 minutes to max 3mg. Child 0.02 mg/kg.
  • If rhythm is restored, follow appropriate protocols e.g., fibrillation, bradycardia, hypotension, etc. If asystole persists, consider termination of efforts.
  • Pulseless Electrical Activity
  • CPR for 2 mins.
  • EKG paddles or leads. Determine presence of PEA (electrical rhythm without pulses).
  • Continue CPR for 2 mins
  • I.V. volume challenge with 300-500 cc. balanced salt solution
  • ET tube, 100 O2
  • Pulseless Electrical Activity (cont.)
  • If bradycardia, Atropine 1 mg I.V., may repeat q 3-5 minutes to max 3mg.
  • If witnessed event, consider Transcutaneous Pacemaker
  • Continuously monitor effectiveness of CPR and oxygenation
  • Use of vasopressor
  • Use of amiodarone vs. lidocaine vs. magnesium
  • Consider Atropine 1 mg for Slow PEA or Asystole
  • When youre tired?
  • After 20 minutes?
  • Dopamine dosing
  • Guidelines 2000 Dopamine 5-20 µg/kg/min
  • Guidelines 2005 Dopamine 2-10 µg/kg/min
  • NO explanation in text. Why
  • Consultation with editors/authors
  • For simplification we decided to have a single dose for dopamine and epinephrine ?????
  • (µg/kg/min vs. µg/min)
  • BUT does it make sense
  • Does the change make sense
  • No new data. The choice is editorial
  • Pro Simpler
  • Con Change for the sake of change
  • Consensus strikes again
  • The skilled provider can start at the appropriate dose
  • Is amiodarone the ONLY drug for WCT?
  • Procainamide, sotolol, ibutilide, lidocaine and magnesium are discussed in the text.
  • The intent is to provide a BASIC framework to manage the majority of situations
  • In the complex patient, the intent is to allow experts to tailor the treatment to the patient and situation.
  • A patient on amiodarone with VT due to thyrotoxicosis induced by amiodarone should not receive more amiodarone
  • Tachydysrhythmias
  • Ventricular Tachycardia - (stable) with adequate perfusion.
  • O2 High flow.
  • 12 Lead prn (prior to and after treatment/conversion))
  • Amiodarone 150 mg over 10 minutes Repeat 150 mg after 10 minutes if VT recurs
  • IF VT persists, cardiovert as in unstable VT.
  • If VT refractory, or Torsade or TCA OD, Magnesium Sulfate 2 grams I.V. slow (5-20 min. dilute in 50-100 cc D5W).
  • Tachydysrhythmias (cont.)
  • Ventricular Tachycardia - (unstable- hypotension, CHF, chest pain, SOB)
  • O2 high flow.
  • Versed 2.5-5 mg I.V. as needed.
  • Synch cardiovert 100, 200, 300, 360j monophasic, OR 100, 150, 200, 200 j biphasic) Peds 0.5 j/kg, 1 j/kg prn
  • If recurrent VT or persistent
  • Amiodarone, per protocol above
  • Magnesium Sulfate, per protocol above
  • Wide-Complex Tachycardia (WCT) of uncertain type (SVT vs. VT)
  • O2 high flow
  • Amiodarone 150 mg bolus over 10 minutes repeat q 10 mins prn
  • If WCT rhythm persists, cardiovert as needed as if unstable.
  • UNSTABLE (see definition above)
  • Cardiovert as per unstable V.T.
  • Supraventricular (Narrow Complex) Tachycardia
  • ATRIAL FIB/FLUTTER NO HYPOTENSION w/ rapid ventricular rate
  • I.V 12 Lead as in above stable rhythms
  • Diltiazem 0.25 mg/kg (maximum 20 mg) given slow over 2 mins. Avoid in patients taking B-blockers
  • After 15 mins. may repeat at 0.35 mg/kg (maximum 25 mg)
  • If conversion, 10mg/hr drip
  • If hypotension after administration, fluids as appropriate and Calcium 250 mg
  • ATRIAL FIBRILLATION/FLUTTER - unstable with rapid ventricular rate
  • Cardiovert synchronized at 100,200,300,360 J. monophasic 100, 150, 200, 200j biphasic
  • Peds 0.5 j/kg, 1 j/kg prn
  • (Versed sedation as needed)
  • Supraventricular Tachycardia (cont.)
  • PSVT, nodal tachycardia, PAT etc.
  • If stable, attempt vagal maneuvers
  • Cough, valsalva
  • Carotid sinus massage
  • Establish bilateral carotid, no bruits do right CSM, wait 1 minute do left CSM if right unsuccessful.
  • If PSVT persists Adenocard
  • Contraindication 2?block/3?block, allergy, KNOWN WPW.
  • Caution asthma, pregnancy, and pt. on Tegretol/Dipyridamole
  • Ineffective in A-fib/A-flutter
  • If PSVT persists stable, i.e. NO hypotension w/ rapid ventricular rate
  • Diltiazem 0.25 mg/kg (maximum 20 mg) given slow over 2 mins. Avoid in patients taking B-blockers after 15 mins. may repeat at 0.35 mg/kg (maximum 25 m
  • If hypotension after administration administer fluids as appropriate and Calcium 250 mg
  • If hypotensive, fluid challenge 200-300 cc balanced salt.
  • Contraindication pulmonary edema
  • If Unstable
  • synchronized Cardioversion at 100, 200, 300, 360j monophasic 50, 100, 150, 200 biphasic
  • Peds 0.5j/kg, 1j/kg prn
  • Versed sedation, as needed
  • Vasopressin
  • Positive epy effects (augment CPP) without epy side effects
  • 1 dose 40 unit to replace first or second dose of epinephrine
  • Studies show most effectiveness in Asystole, PEA and VF/VT no pulse.
  • Higher BP and HR post arrest if used
  • Can be given ET ( not preferred)
  • Handicapped by extreme difficulty in giving for emergencies
  • Most Effective in cardiac arrest with heart history
  • Only 2 doses in cardiac arrest (300mg, 150mg)
  • Must ALWAYS be diluted
  • DO NOT administer with other drugs that prolong QT
  • Cannot go down the tube
  • 150mg over 10 minutes every 10 minutes as needed
  • Maint. Drip _at_ 1 mg/min
  • Relieves bradycardia,
  • Ineffective if bradycardia is from
  • 1 mg for patients without pulse
  • 0.5mg for patients with a pulse
  • Effective in cardiac arrest with NO heart history
  • Increases the electrical energy required to defibrillate by more than 50
  • Used if Amiodarone is contraindicated
  • Can be given by ET
  • Must be diluted
  • Given over 5-20 minutes in cardiac arrest
  • 5- 60 minutes in Torsades with a pulse
  • Life threatening ventricular arrythmias due to digitalis toxicity
  • Tricyclic OD, Torsades, hypomagnesemia
  • Caution in renal failure patients
  • ET drugs discouraged
  • IO or IV preferred
  • TX Betablocker and Ca Channel Toxicity
  • 6, 12, 12 mg stable patient
  • 12,12,18 for stable patient on theophyllin or caffeine OD
  • 3 mg for dipyridamole (persantine), tegretol, heart transplant (denervated heart)
  • Monophasic versus Biphasic
  • Optimize tissue perfusion, especially for the brain
  • Induction of mild hypothermia (33-34 C for 12-24 hours
  • During resuscitation spontaneous cooling occurs to 35 C
  • Improves neurologic recovery
  • Low blood pressure within 2 hours
  • Associated with poor neuro outcomes

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Slide Set | 2020 AHA/ACC Guideline for the Management of Patients With Valvular Heart Disease

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See Also: Guideline Hub | Valvular Heart Disease

Date: December 17, 2020    

Keywords: Anticoagulants, Fibrinolytic Agents, Platelet Aggregation Inhibitors, Aortic Valve Insufficiency, Aortic Valve Stenosis, Aortic Valve, Aortic Diseases, Perioperative Period, Cardiac Catheterization, Cardiac Imaging Techniques, Magnetic Resonance Imaging, Cardiovascular Surgical Procedures, Diagnostic Techniques, Cardiovascular, Drug Therapy, Echocardiography, Endocarditis, Exercise Test, Diagnostic Imaging, Angiography, Hemodynamics, Mitral Valve Stenosis, Mitral Valve Insufficiency, Heart Murmurs, Mitral Valve, Pregnancy, Heart Valve Prosthesis, Decision Making, Heart Valve Diseases, Heart Defects, Congenital, Patient Care Team, Thromboembolism, Thrombosis, Transcatheter Aortic Valve Replacement, Heart Valve Prosthesis Implantation, Catheters, Echocardiography, Transesophageal, Tricuspid Valve

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  1. Advanced Cardiac Life Support

    39 END. Download ppt "Advanced Cardiac Life Support". PRINCIPLES OF MANAGEMENT Excellent basic life support & its importance Excellent CPR & early defibrillation for treatable arrhythmias remain the cornerstones of basic & ACLS. Although the 2010 American Heart Association (AHA) Guidelines for ACLS suggest several revisions, including ...

  2. PDF ACLS Megacode Testing Scenarios

    Adult Bradycardia Algorithm. Instructor notes: His vital signs include heart rate 30/min, respiratory rate 16/min, blood pressure 80/48 mm Hg, Spo2 98% on 3 L by nasal prongs, temperature 36.5°C, and blood glucose 195 mg/dL (10.8 mmol/L). A rhythm strip shows wide QRS ventricular escape rhythm at 30, with a long QT.

  3. PDF Highlights of the 2020 American Heart Association's Guidelines for CPR

    Introduction. These Highlights summarize the key issues and changes in the 2020 American Heart Association (AHA) Guidelines for Cardiopulmonary Resuscitation (CPR) and Emergency Cardiovascular Care (ECC). The 2020 Guidelines are a comprehensive revision of the AHA's guidelines for adult, pediatric, neonatal, resuscitation education science ...

  4. PPT

    Presentation Transcript. ASP Medical Clinic/ sept 2012. ACLS Brief Overview & 2010 AHA ECC Guidelines Dr.M.Hajikarimi interventional cardiologist. WHAT IS ACLS? • Advanced Cardiac Life Support • An extension of BLS (Basic Life Support) • Implementation of advanced life support is not intended to suggest an abrupt cessation of basic life ...

  5. ACLS

    Advanced Cardiovascular Life Support (ACLS) The AHA's ACLS course builds on the foundation of lifesaving BLS skills, emphasizing the importance of continuous, high-quality CPR. Reflects science and education from the American Heart Association Guidelines Update for CPR and Emergency Cardiovascular Care (ECC).

  6. PPT

    American Heart Association (AHA)u2019s Advanced Cardiovascular Life Support (ACLS) Course has been updated to reflect new science in the American Heart Association Guidelines Update for CPR and Emergency Cardiovascular Care. This course builds on the foundation of lifesaving BLS skills, emphasizing the importance of continuous, high-quality CPR. Advanced Cardiovascular Life Support (ACLS) is ...

  7. ACLS

    ACLS.pptx - Free download as Powerpoint Presentation (.ppt / .pptx), PDF File (.pdf), Text File (.txt) or view presentation slides online. This document provides guidelines from the 2015 American Heart Association for cardiopulmonary resuscitation (CPR) and emergency cardiovascular care. It discusses the importance of basic life support (BLS) as the foundation for saving lives after cardiac ...

  8. ACLS

    ACLS PPT - Free download as Powerpoint Presentation (.ppt / .pptx), PDF File (.pdf), Text File (.txt) or view presentation slides online. The document outlines guidelines for adult advanced cardiovascular life support, including the importance of high-quality CPR, use of adjuncts like supplemental oxygen and advanced airways, defibrillating shockable rhythms like ventricular fibrillation, and ...

  9. Slide Set

    Slide Set | 2023 ACC/AHA/ACCP/HRS Guideline for the Diagnosis and Management of Atrial Fibrillation . Print; Download PowerPoint File. Description: A Report of the American College of Cardiology/American Heart Association Joint Committee on Clinical Practice Guidelines. See Also: Guideline Hub | Atrial Fibrillation.

  10. PDF Acls Powerpoint Presentation (2024)

    ACLS for Experienced Providers Manual and Resource Text American Heart Association Staff,2017-06-07 Product 15-1064 ACLS, CPR, and PALS Shirley A. Jones,2014-03-24 Here's all the information you need to respond to cardiac emergencies in one pocket-

  11. Part 3: Adult Basic and Advanced Life Support: 2020 ...

    In 2015, approximately 350 000 adults in the United States experienced nontraumatic out-of-hospital cardiac arrest (OHCA) attended by emergency medical services (EMS) personnel. 1 Approximately 10.4% of patients with OHCA survive their initial hospitalization, and 8.2% survive with good functional status. The key drivers of successful resuscitation from OHCA are lay rescuer cardiopulmonary ...

  12. Advanced Cardiac Life Support PowerPoint Presentation

    Free Download Advanced Cardiac Life Support PowerPoint Presentation. Check out this medical presentation on Emergency Medical Services (EMS), which is titled "Advanced Cardiac Life Support", to know about Advanced Cardiac Life Support. ... ACLS Algorithm Primary Survey Shock - 360 J Secondary Survey Vasopressor - Epi or Vasopressin IV Shock ...

  13. Slide Set

    Download PowerPoint File. Description: Slide Set | 2018 ACC/AHA/HRS Guideline on the Evaluation and Management of Patients With Bradycardia and Cardiac Conduction Delay Date: November 28, 2018

  14. PPT

    Presentation Transcript. Advanced Cardiac Life Support (ACLS) By: Diana Blum MSN Metropolitan Community College Nursing 2150. STABLE • These patients generally have an EKG rhythm that is undesirable. • their vitals signs are stable • they have no complaints such as, shortness of breath, chest pain or confusion. • if rhythm untreated the ...

  15. AHA Guidelines ACLS

    retesting for improper performance. Rapid rate with full chest recoil is emphasized. Rate of 302 compression to ventilation with BVM. 100 compressions unsynchronized to 8-10. ventilations controlled airway. 15. Compressions. Research indicates- 2 minutes of CPR prior to. first defibrillation for a non witnessed cardiac.

  16. Slide Set

    Download PowerPoint File. Description: See Also: Guideline Hub | Valvular Heart Disease. Date: December 17, 2020 Keywords: Anticoagulants, Fibrinolytic Agents ...